As with our patient, when large portions of the cranium have been removed by operation, external injury, or exfoliation, the brain is deprived of its support, and, by the pulsation of its arteries, the dura matter is rubbed against the rough edges of the bone, by which it ulcerates, and the brain then protrudes. This protrusion from the cerebrum is more particularly apt to occur when the dura matter has been wounded by accident or design and it may present itself under three different forms: It may consist either of disorganized pure cerebral matter, partly of cerebral matter and of coagulated blood; or be an organized mass.
The first generally occurs immediately after the injury, and arises from the substance of the brain being wounded, and a portion of it being separated from the common mass.
The second most commonly takes place some days after the injury, and seems to arise from the rupture of one or more blood vessels a little below the surface of the brain, in consequence of which, a portion of this viscus is mechanically raised through the opening in the dura matter, skull, and integuments. This protrusion is of a dark color and is at first covered by the pia matter, but the internal hemorrhage continuing, the brain is still farther raised. This membrane then gives way. At length, the the brain yields and the blood is effused upon the surface of the tumor.
The third species of protrusion from the brain follows abscess or ulceration of that organ, hence does not appear for many days, and is as completely organized as the fungi springing from other ulcers. The fungus which arises from the brain shoots up with great rapidity, is of a greyish colour, and is generally about the size of a pigeon’s head, but, on some occasions, it is as large as a hen’s egg or a small orange. Its appearance is always preceded by an opening in the dura matter and is always connected with ulceration of the brain.
External herniations, also known as fungus cerbrei, are the rarest of the acquired herniations. They are most frequently caused by post surgical and post traumatic defects that allow swollen or displaced brain to pass through. This type of herniation may be beneficial by relieving intracranial pressure and thereby preventing more devastating complications due to internal herniation; however; external exposure of the brain can result in infections, complications, and many of these patients have a poor prognosis because of the severity of their underlying illness. Both CT and MR imaging are effective at demonstrating these herniations. MR imaging, however, is clearly superior at defining soft tissue, which can be critical in certain clinical scenarios. Distingushing brain tissue from other tissues such as granulation tissue, cholesteatoma, tumor, or other lesions involving defects of the skull is essential and MR imaging accomplishes this well.




Observation
Clinical History: bleed, follow up
Technique: CT scan of the head without intravenous contrast
Compared with prior study dated January 30, 2010
The presence of streak artifact limits the study. There is interval development of some degree of herniation of the left frontal lobe through the left craniectomy defect with interval development of subdural hygroma in the right frontoparietal convexity and evolution of the previously noted mild left to right subfalcine herniation into a mild degree of right to left subfalcine herniation. There is partial interval improvement of the pneumocephalus in the left frontal and left parietal convexities. There is no significant interval change in the left frontotemporoparietal, parafalcine, and left paratentorial hyperdensities, which may reflect subdural hemorrhage. There is interval development of opacities in some of the ethmoid air cells and sphenoid sinuses.
Impression
INTERVAL DEVELOPMENT OF SOME DEGREE OF HERNIATION OF THE LEFT FRONTAL LOBE THROUGH THE LEFT CRANIECTOMY DEFECT. CONSISTENT WITH DEVELOPMENT OF POST-SURGICAL PSEUDOENCEPHALOCELE (FUNGUS CEREBRI). ASSOCIATED INTERVAL DEVELOPMENT OF RIGHT-SIDED SUBDURAL HYGROMA AND MILD RIGHT TO LEFT SHIFT. NO SIGNIFICANT INTERVAL CHANGE IN THE LEFT FRONTOTEMPOROPARIETAL, PARAFALCINE, AND LEFT PARATENTORIAL HYPERDENSITIES, MAY REFLECT SUBDURAL HEMORRHAGE. INTERVAL DEVELOPMENT OF OPACITIES IN THE ETHMOID AND SPHENOID SINUSES, SUGGESTIVE OF SINUSITIS. PARTIAL INTERVAL IMPROVEMENT OF THE PNEUMOCEPHALUS IN THE LEFT FRONTAL AND LEFT PARIETAL CONVEXITIES.